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A diuretic (furosemide, torsemide, bumetanide) that removes fluid in CKD and dialysis patients by blocking sodium reabsorption.
Loop diuretics act on the ascending loop of Henle and remain effective at low eGFR (unlike thiazides). They manage fluid overload, edema, and hypertension in advanced CKD; in dialysis, they help maintain residual urine output. Monitoring includes potassium (often low — hypokalemia), sodium, and kidney function. High doses may be needed as CKD progresses.
Excess extracellular fluid volume in kidney patients from inadequate excretion, causing edema, hypertension, pulmonary congestion, and increased cardiovascular risk.
Blood potassium below ~3.5 mEq/L - less common than hyperkalemia in CKD but dangerous, causing muscle weakness and arrhythmias.
Persistently elevated blood pressure - the second leading cause of kidney failure and a driver of CKD progression at every stage.
The target body weight of a dialysis patient after all excess fluid has been removed — the weight at which blood pressure is normal without medication.
A sudden drop in kidney function over hours to days - reversible if caught early, but a major risk factor for chronic kidney disease.
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This content is a general reference, not medical advice, a diagnosis, or a treatment plan. Do not change your diet, fluids, medicines, or dialysis plan without your nephrologist or renal dietitian. Individual recommendations depend on your labs, medications, conditions, and care plan.