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A scarring pattern of the glomeruli that causes nephrotic-range proteinuria and progressive CKD.
FSGS can be primary (podocyte injury) or secondary (obesity, HIV, reflux, drugs). It often presents with heavy proteinuria, edema, and hypertension, and carries a high risk of progression. Treatment includes RAAS blockade, steroids/immunosuppression in primary disease, and managing the secondary cause. Response to treatment predicts long-term kidney survival.
Heavy protein loss in urine (proteinuria >3.5 g/day) with low albumin, edema, and high cholesterol - a hallmark of glomerular disease.
Abnormal presence of protein in the urine, a key marker of kidney damage and the strongest predictor of CKD progression and cardiovascular risk.
Inflammation of the kidney's filtering units (glomeruli) - a diverse family of diseases often presenting with blood and protein in urine.
Albumin leaking into urine — the earliest and most important marker of kidney filter damage, measured by urine albumin-to-creatinine ratio (UACR).
A procedure that takes a tiny sample of kidney tissue for a microscope to diagnose glomerular disease, guide treatment, and assess injury or rejection.
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This content is a general reference, not medical advice, a diagnosis, or a treatment plan. Do not change your diet, fluids, medicines, or dialysis plan without your nephrologist or renal dietitian. Individual recommendations depend on your labs, medications, conditions, and care plan.