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Distinguish prerenal azotemia from intrinsic kidney disease. Updated 2026-08.
Distinguish prerenal azotemia from intrinsic kidney disease
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For education and screening only — not a substitute for professional medical assessment. Results should be interpreted by your healthcare provider.
Suggests prerenal azotemia: dehydration, vomiting/diarrhea, heart failure, or reduced kidney perfusion. Recheck after fluid repletion; if it normalizes, the kidneys were intact.
Typical ratio. Both BUN and creatinine may still be elevated together in intrinsic kidney disease — interpret with eGFR and clinical history.
Low ratio — seen in intrinsic renal disease (ATN), low protein intake, malnutrition, or liver disease. Correlate with the clinical picture.
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This content is a general reference, not medical advice, a diagnosis, or a treatment plan. Do not change your diet, fluids, medicines, or dialysis plan without your nephrologist or renal dietitian. Individual recommendations depend on your labs, medications, conditions, and care plan.